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In addition, in vitro treatment with SAM or MTA promoted the expression of exhaustion marks, inhibited IFN-γ expression, and caused a closed chromatin structure in human CD8+ T cells ( MAT2A knockout protected tumor-infiltrating CD8+ T cells from exhaustion in a syngeneic mouse tumor model ( Figure 2 Perspective on methionine metabolism in T cell immunotherapy Improving cancer immunotherapy by targeting the methionine cycle of T cells is an area that remains unexplored
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doi: 10.7554/eLife.27014.040 189 SolovyevM
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