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In silico analysis evidenced that SSTR2 was a putative target of miR-185 and, intriguingly, miR-185 was upregulated in tumor samples from SSA non-responder adenoma patients with respect to controls while it was downregulated in the SSA responder patients ( MiR-338-3p inhibition reduced both GH mRNA and protein levels in GH3 cells and reduced cell proliferation (62)
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