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82 Lack of phosphatidylethanolamine (PE) methylation leads to the SAM accumulation, which results in hypermethylation of histones and the major phosphatase PP2A, dependency on cysteine, and sensitivity to oxidative stress

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Reduced FXR expression or altered activity contributes to the pathogenesis of IBD, CRC, and GEAC (89, 90), positioning FXR as a potential tumor suppressor in the gastrointestinal tract (90)