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In the pathophysiology of arterial hypertension, AT-II binds to angiotensin II type 1 (AT1) receptors, which leads to G-protein conjugation and causes signaling via secondary messengers, including diacylglycerol, which, as a strong endogenous activator of protein kinase C, contributes to the activation of phagocytic NADPH oxidase by p47phox [24]

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Vitamin D and Autoimmune Diseases: Is Vitamin D Receptor (VDR) Polymorphism the Culprit