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Structure and mechanism of mammalian thioredoxin reductase: The active site is a redox-active selenolthiol/selenenylsulfide formed from the conserved cysteine-selenocysteine sequence
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Despite this, the evidence suggests that immunological dysregulation, inflammation, oxidative stress, mitochondrial dysfunction, and excitotoxicity are key components in ASD pathogenesis (3, 10, 20, 21)
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